Internet Marketing Workshop - Steve Mills Marketing

The Canada-UK Chamber of Commerce invites you to a half-day ?

?Internet Marketing Made Simple 2013?

with Keynote Speaker?

Steve Mills ? The Prudent Marketer

Founder of The LinkedIn Academy?& the Marketing without Money Programme

During the workshop Steve will discuss

• Drive over 1,000 people per month to your website from LinkedIn
• Dominate the first page of Google (not just be on it)
• Use YouTube the world?s 2nd biggest search engine (there are currently 4 billion videos watched on YouTube every day)
• Convert more of your website visits into enquirers by utilising?lead conversion technology?(it is simpler than it sounds)
• Improve your return on investment from your PPC Advertising and your website
• Get hundreds of people telling others how great you are on Google, YouTube, LinkedIn, Bogger.com, Facebook and even other peoples blogs
• Win more business, more easily, more often

Tuesday 4 June 2013

The Large Salon Room,?Canadian High Commission, Macdonald House,?41 Grosvenor St, London, W1K 4AA

Registration & coffees etc: 09.00 ? 09.30 Half-day Workshop: 09.30 ? 13.00

Members & their guests: ?85inc VAT Non Members: ?110inc VAT

Sponsored by:

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To attend this event please RSVP with payment & registration form where appropriate by Monday 3 June 2013 to: events@canada-uk.org? T: +44 (0) 20 7258 6578

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Steve Mills is known as ?The Prudent Marketer? and provides Marketing Advice, Marketing Training, Social Media training including Facebook, LinkedIn, Twitter etc, for new and small businesses.?He is the author of four books on marketing.?Steve helps businesses to make money without spending money: he specialises in ?Marketing without Money strategies? and helps clients to market themselves using many of these techniques and strategies.

A leading Marketing Expert and Professional Sales trainer with over 25 years? experience, he is the founder of The LinkedIn Training Academy and has also developed an online training programme designed to teach business owners exactly how to use LinkedIn effectively. The LinkedIn Academy is privately run and is not owned, or endorsed by LinkedIn plc.

Over? the past 17 years Steve has helped businesses develop systems and strategies to:

• Improve the ROI from their current marketing
• Measure and track everything
• Test their marketing to get the best possible results from every marketing pound
• Make a marketing plan and set a budget with specific targets
• Make full use of referrals and with a plan and scripts for winning them
• Systemise their marketing and sales processes

Steve has appeared on many tv programmes on the BBC and ITV including: Blue Peter, Wish You Were Here, Look North, and South Today. And multiple commercial radio stations including: BBC Radio Berkshire, Oxfordshire, Sheffield, Jersey, Leicester, Hampshire, Jersey, and Leeds.

He has been featured in Business Today, The Sun, The Star, The News of the World, Success Magazine, The Newbury Weekly News, Reading Chronicle, Berkshire Life, and multiple on-line websites.

Steve is a former successful international table tennis player and fully understands the value of having a good coach. When he went into business for himself he applied many of the principles he learned as a sportsman including a continual and never ending journey to be the best he could be. He now inspires the clients he works for with the same ethos.

?I really care about people. I have a great sense of humour and I really want to make a difference by sharing with others what I do best.?I am committed to consistent and never ending improvement both personally and for those I work with.?

Source: http://www.stevemillsmarketing.com/canadian-chamber-of-commerce-internet-marketing-workshop/

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Spearmint Tea - YumSugar

For a refreshing drink with a minty twist, try this easy recipe for spearmint sun tea by Food Fashion Fun.

Once you've brewed your tea, simple additions like mint and lemon infuse the tea with summertime flavor.

For more ? and the recipe ? visit her blog, and then be sure to share your food photos via Savory Sights on POPSUGAR Social or by starting your own blog. If you're on Instagram, then chime in on the conversation with the hashtag #savorysight.

Source: http://www.yumsugar.com/Spearmint-Tea-30444473

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Student Opinion | Would You Want a Pair of Google's Computer ...

Are wearable computers the next big technological innovation? Do you see yourself walking around with computerized glasses a few years from now, snapping pictures, sending messages or getting directions? Or is the hype and controversy misplaced?

Would you want the device, known as Google Glass?

In the article ?Google Glass Picks Up Early Signal: Keep Out,? David Streitfeld writes about the resistance to Google?s wearable technology, which is still a work in progress.

Google?s wearable computer, the most anticipated piece of electronic wizardry since the iPad and iPhone, will not go on sale for many months.

But the resistance is already under way.

The glasseslike device, which allows users to access the Internet, take photos and film short snippets, has been pre-emptively banned by a Seattle bar. Large parts of Las Vegas will not welcome wearers. West Virginia legislators tried to make it illegal to use the gadget, known as Google Glass, while driving.

?This is just the beginning,? said Timothy Toohey, a Los Angeles lawyer specializing in privacy issues. ?Google Glass is going to cause quite a brawl.?

As personal technology becomes increasingly nimble and invisible, Glass is prompting questions of whether it will distract drivers, upend relationships and strip people of what little privacy they still have in public.

Students: Tell us ?

• Would you want a pair of Google?s computer glasses?
• Are you an early adopter? Do you like to own the newest gadgets right when they come out?
• What would you use a wearable computer like Google Glass for?
• Do you think Google Glass takes technology too far? Will it invade our privacy when people can so easily shoot videos or record conversations secretly? Will it distract drivers ? or students in the classroom?
• Do you think smart glasses will be the next big must-have device, like iPhones and iPads, or do you think they will never attract such mass appeal?

Students 13 and older are invited to comment below. Please use only your first name. For privacy policy reasons, we will not publish student comments that include a last name.

Source: http://learning.blogs.nytimes.com/2013/05/08/would-you-want-a-pair-of-google-glass/

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CEOAS - WR Seminar - Christopher Duffy, Pennsylvania State University

CEOAS - WR Seminar - Christopher Duffy, Pennsylvania State University

"Measure and model the isotopic ?age? of water in hydroecological systems"

Wednesday, May 8, 2013 4:00 PM - 5:00 PM

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Campus Location:
Room:	4000
Contact:	Biana Weatherford
Contact Email:	bweatherford at coas.oregonstate.edu
Contact Department:	Earth, Ocean & Atmo Sci

This event appears on the following calendars:

Source: http://calendar.oregonstate.edu/event/81430/

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Two genes that combine to cause rare syndrome identified

May 8, 2013 ? Researchers from Massachusetts General Hospital (MGH) and Duke University have identified genetic mutations that appear to underlie a rare but devastating syndrome combining reproductive failure with cerebellar ataxia -- a lack of muscle coordination -- and dementia. In a paper that will appear in the May 23 New England Journal of Medicine and is receiving early online release, the investigators describe finding mutations in one or both of two genes involved in a cellular process called ubiquitination in affected members of five unrelated families.

"This study highlights, for the first time, the importance of the ubiquitin system in a syndrome characterized by ataxia and hypogonadotropic hypogonadism -- reproductive failure due to abnormal signaling from the brain or pituitary gland," says Stephanie Seminara, MD, of the Reproductive Endocrine Unit in the MGH Department of Medicine, co-senior author of the report. "It also demonstrates how combining robust genomics with detailed functional assays can unlock complex genetic architecture."

Caused by lesions in the part of the brain responsible for coordination and balance, cerebellar ataxia can begin with difficulty walking or speaking and progress to complete disability of those functions. Genes associated with several syndromes characterized by ataxia have been identified, but none had previously been associated with the rare combination of ataxia and reproductive failure, which was first described more than 100 years ago. Several such patients have been referred to the MGH Reproductive Endocrine Unit, including a Palestinian family with several affected members who also developed dementia.

Seminara notes that, while ataxia and hypogonadotropic hypogonadism each may have several possible genetic causes, the combination of both conditions is so rare that it is more likely to be caused by mutations in a particular gene or related genes. In collaboration with researchers from the Center for Human Disease Modeling at Duke -- directed by Nicholas Katsanis, PhD, co-senior author of the NEJM article -- her team conducted whole-exome sequencing of DNA from an affected member of the Palestinian family. That screening found rare variants in both copies of 13 genes, and two of those variants were also found in samples from the patient's two affected siblings but not in several unaffected family members.

Both of the mutated genes are involved in ubiquitination, a process by which cellular proteins are marked for degredation by a protein called ubiquitin. One of them, RNF216, codes for an enzyme that attaches ubiquitin to the protein; the other, OTUD4, codes for a protein that removes ubiquitin. The researchers then sequenced both of these proteins in samples from an additional nine affected individuals from seven different families. They found that one of those individuals had two different RNF216 mutations, four others -- two in the same family -- had mutations in a single copy of that gene, but none had mutated versions of OTUD4.

All of the individuals with RNF216 mutations had similar medical histories, characterized by a lack of normal hormonal secretion, progressive ataxia and dementia; and all of those with mutations in both genes died in their 30s or 40s. Neuroimaging studies revealed similar brain abnormalities -- including atrophy of the cerebellum and cortex -- in individuals with RNF216 mutations. The four studied individuals without RNF216 mutations had very different histories, with less severe symptoms.

To get a better idea of the functional consequences of mutations in these two genes, the researchers disrupted their expression in zebrafish and found that blocking either RNF216 or OTUD4 caused disorganization of the cerebellum and reduced the size of eyes and a portion of the midbrain. The abnormalities were even greater when both genes were blocked but could be eliminated if production of the relevant proteins was induced by the introduction of the corresponding human RNA.

"The presence of RNF216 mutations in several families made its role in causing this syndrome clear, but finding OTUD4 mutation in only one family raised the question of whether it actually contributed to the disease or was just an 'innocent bystander'," says Katsanis. "The zebrafish work provided critical evidence that both genes function in a common pathway, since blocking either of them produced similar effects. And the fact that blocking both genes had a synergistic effect lends further evidence to the two genes' operating in the same pathway and to the contribution of OTUD4 mutations to this syndrome."

Although exactly how these mutations lead to the symptoms seen in these individuals is unknown, the researchers note that identifying these genes may someday lead to therapies -- potentially including drugs currently being developed for other disorders involving ubiquitination, including Parkinson's disease -- and enable genetic screening and counseling for affected families. They also hope to investigate whether less severe mutations in these genes may contribute to the presence of ataxia, dementia or hypogonadism in isolation.

Seminara is an assistant professor of Medicine at Harvard Medical School, and Katsanis is the Jean and George W. Brumley Professor of Cell Biology and Pediatrics at Duke University Medical Center. The co-lead authors of the NEJM report are David Margonin, MD, PhD, MGH Department of Neurology; Maria Kousi, PhD, Duke Center for Human Disease Modeling; and Yee-Ming Chan, MD, PhD, MGH Reproductive Endocrine Unit and Boston Children's Hospital. The study was supported by National Institute for Child Health and Human Development grants R01 HD043341, R01 HD042601 and U54 HD028138 and by other National Institutes of Health grants.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/health_medicine/genes/~3/SGGyNlcd_wo/130508213102.htm

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Raw live results: May 6, 2013

All WWE programming, talent names, images, likenesses, slogans, wrestling moves, trademarks, logos and copyrights are the exclusive property of WWE, Inc. and its subsidiaries. All other trademarks, logos and copyrights are the property of their respective owners. ? 2013 WWE, Inc. All Rights Reserved. This website is based in the United States. By submitting personal information to this website you consent to your information being maintained in the U.S., subject to applicable U.S. laws. U.S. law may be different than the law of your home country. WrestleMania XXIX (NY/NJ) logo TM & ? 2013 WWE. All Rights Reserved. The Empire State Building design is a registered trademark and used with permission by ESBC.

Source: http://www.wwe.com/shows/raw/2013-05-06/wwe-raw-results

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Commands from the matrix: Cellular environment controls formation and activity of neuronal connections

May 6, 2013 ? Environment moulds behaviour -- and not just that of people in society, but also at the microscopic level. This is because, for their function, neurons are dependent on the cell environment, the so-termed extracellular matrix. Researchers at the Ruhr-Universit?t have found evidence that this complex network of molecules controls the formation and activity of the neuronal connections.

The team led by Dr. Maren Gei?ler und Prof. Andreas Faissner from the Department of Cell Morphology and Molecular Neurobiology reports in the Journal of Neuroscience in collaboration with the team of Dr. Ainhara Aguado, Prof. Christian Wetzel and Prof. Hanns Hatt from the Department of Cell Physiology.

Neurons and astrocytes in culture

In cooperation with Prof. Uwe Rauch from Lund University in Sweden, Bochum's biologists examined cells from the brains of two mouse species: a species with a normal extracellular matrix and a species which lacked four components of the extracellular matrix due to genetic manipulation, namely the molecules tenascin-C, tenascin-R, neurocan and brevican. They took the cells from the hippocampus, a brain structure that is crucial for the long-term memory. The team not only examined neurons but also astrocytes, which are in close contact with the neurons, support their function and secrete molecules for the extracellular matrix.

Formation, stability and activity of the neuronal connections depend on the matrix

The researchers cultivated the neurons and astrocytes together for four weeks with a specially developed culture strategy. Among other things, they observed how many connections, known as synapses, the neurons formed with each other and how stable these were over time. If either the astrocytes or the neurons in the culture dish derived from animals with a reduced extracellular matrix, these synapses proved to be less stable in the medium term, and their number was significantly reduced. Together with the Department of Cell Physiology at the RUB and the University of Regensburg, the team also showed that the neurons with a mutated matrix showed lower spontaneous activity than normal cells. The extracellular matrix thus regulates the formation, stability and activity of the neuronal connections. The researchers also examined a special structure of the extracellular matrix, the so-called perineuronal nets, which the Nobel laureate Camillo Golgi first described more than a century ago. They were significantly reduced in the environment of genetically modified cells.

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Story Source:

The above story is reprinted from materials provided by Ruhr-Universitaet-Bochum.

Note: Materials may be edited for content and length. For further information, please contact the source cited above.

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Journal Reference:

1. M. Geissler, C. Gottschling, A. Aguado, U. Rauch, C. H. Wetzel, H. Hatt, A. Faissner. Primary Hippocampal Neurons, Which Lack Four Crucial Extracellular Matrix Molecules, Display Abnormalities of Synaptic Structure and Function and Severe Deficits in Perineuronal Net Formation. Journal of Neuroscience, 2013; 33 (18): 7742 DOI: 10.1523/JNEUROSCI.3275-12.2013

Note: If no author is given, the source is cited instead.

Disclaimer: This article is not intended to provide medical advice, diagnosis or treatment. Views expressed here do not necessarily reflect those of ScienceDaily or its staff.

Source: http://feeds.sciencedaily.com/~r/sciencedaily/~3/wiWyf_xhKbY/130506102928.htm

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